MDS Abstracts

Abstracts from the International Congress of Parkinson’s and Movement Disorders.

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Articles tagged "Mitochondria"

  • 2017 International Congress

    Impaired stress-induced mitophagy in parkinsonian LRRK2(R1441G) knockin mutant mice

    P.W.-l. Ho, C.S.-c. Lam, H.-f. Liu, G.C.-t. Leung, L. Li, M.H.-w. Kung, D. Ramsden, S.-l. Ho (Hong Kong, Hong Kong)

    Objective: 1. Morphological changes and accumulation of ubiquitinated mitochondria in the striatum of aged leucine-rich-repeat kinase 2 (LRRK2)R1441G knockin mutant mice (Liu et al., 2014,…
  • 2017 International Congress

    Citrus extract A protects dopaminergic neuron by modulating mitochondrial respiration and reactive oxygen species

    Y. Jang, J. Han, S.J. Kim, M.J. Lee, I. Ryu, X. Ju, H. Chu, M.J. Ryu, E.S. Oh, S.-Y. Choi, W. Chung, J.Y. Heo, G.R. Kweon (Daejeon, Republic of Korea)

    Objective: This study aims to validate the protective effect of citrus extract A (CEA) against loss of dopamine neurons in PD by dual targeting of…
  • 2017 International Congress

    CR6-interacting factor1 Deficiency in Dopamine Neurons Triggers Early-onset Parkinsonism in Mice

    I. Ryu, J. Han, Y. Jang, S.J. Kim, j. Kim, M.J. Lee, X. Ju, M.J. Ryu, S.-Y. Choi, W. Chung, E. Oh, G.R. Kweon (Daejeon, Republic of Korea)

    Objective: One candidate is CR6-interacting factor1 (CRIF1), which controls translation and membrane insertion of 13 mitochondrial proteins involved in oxidative phosphorylation. Here we determined the…
  • 2017 International Congress

    Inhibition of mitochondrial complex I synthesis by chloramphenicol mitigates dopaminergic neuronal cell loss in PQ-induced parkinsonism

    S.J. Kim, J. Han, Y. Jang, J. Kim, M.J. Lee, I. Ryu, X. Ju, M.J. Ryu, W. Chung, J.Y. Heo (Daejeon, Republic of Korea)

    Objective: To find the potent drug for prevention of PD, we screened with above 1000 drugs in the PQ treated cells which currently used drug…
  • 2016 International Congress

    Mild mitochondrial impairment promotes corticostriatal synaptic plasticity alterations in PINK1 heterozygous knockout mice

    P. Imbriani, G. Martella, G. Madeo, M. Maltese, V. Vanni, E. Ferraro, E.M. Valente, T. Schirinzi, L. Bonanni, J. Shen, N.B. Mercuri, P. Bonsi, A. Pisani (Rome, Italy)

    Objective: To investigate the effects of the exposure to low-dose rotenone of heterozygous PINK1 knockout (PINK1+/-) mice, compared to their wild-type littermates (PINK1+/+), on dopamine-dependent…
  • 2016 International Congress

    Enteric neurons reveal substantial in vivo mitochondrial changes in Parkinson’s disease

    P.M.A. Antony, A.S. Baumuratov, M. Ostaszewski, F. He, L. Salamanca, L. Antunes, J. Weber, L. Longhino, P. Derkinderen, R. Balling, W. Koopman, N. Diederich (Belvaux, Luxembourg)

    Objective: Study in vivo mitochondrial and ganglionic morphometrics of enteric neurons in Parkinson's disease. Background: While well established in autopsy studies, the crucial role of…
  • 2016 International Congress

    Evaluation of the role of oxidative stress in Parkinson’s disease and Parkinson plus syndrome

    R. Qadri, M.A. Faiq, M. Behari, V. Goyal, A.K. Mukhopadhyay (New Delhi, India)

    Objective: To evaluate the role of oxidative stress in peripheral blood in patients with Parkinson's disease (PD) and Parkinson plus syndrome (PPS). Background: Slight excess…
  • 2016 International Congress

    Increased cerebrospinal fluid lactate levels in Parkison’s disease: Is it a proof of mitochondrial inefficiency?

    C. Liguori, A. Stefani, E. Olivola, N.B. Mercuri, M. Pierantozzi (Rome, Italy)

    Objective: The aim of the present study was to investigate the impairment of neuronal energy metabolism, as measured by means of cerebrospinal fluid (CSF) lactate…
  • 2016 International Congress

    Beyond initial clinical phenotype of Madelung disease: Myoclonus, gait disorder and multisystem involvement led to genetic diagnosis

    R. López-Blanco, A. Rojo-Sebastián, M.H. Torregrosa-Martínez, M. Molina-Sánchez, A. Blázquez-Encinar, M.Á. Martín-Casanueva (Madrid, Spain)

    Objective: To report the clinical phenotype of an uncommon hereditary systemic disease with slowly progressive gait and movement disorder with genetic identification of two concurrent…
  • 2016 International Congress

    Epigenome-wide association study of Parkinson’s disease

    B. Ritz, Y.H. Chuang, S. Horvath, Y. Bordelon, J. Bronstein (Los Angeles, CA, USA)

    Objective: To identify gene-specific DNA methylation associated with Parkinson's disease (PD). Background: There is a growing interest in the role of epigenetic DNA methylation (DNAm)…
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